Search results for "Steroid 21-Hydroxylase"

showing 9 items of 9 documents

Clinical and hormonal characteristics in heterozygote carriers of congenital adrenal hyperplasia

2020

Abstract Non-classical congenital adrenal hyperplasia (NC-CAH) includes a group of genetic disorders due to a broad class of CYP21A2 variants identifying a disease-causing ‘C’ genotype. The heterozygous carriers of CYP21 mutations are at increased risk of developing clinically evident hyperandrogenism, even though clinical and laboratory characteristics are still underestimated. With the aim of obtaining a more accurate delineation of the phenotype of heterozygous carrier of CAH, we analyzed clinical, biochemical and molecular characteristics in a cohort of Sicilian subjects. Fifty-seven females with biallelic and monoallelic CYP21A2 variants classifying NC-CAH (24) and heterozygous carrier…

0301 basic medicineHirsutismHydrocortisoneendocrine system diseasesEndocrinology Diabetes and MetabolismClinical BiochemistryPhysiologyOverweighturologic and male genital diseasesBiochemistrySettore MED/13 - Endocrinologia0302 clinical medicineEndocrinologySettore BIO/10 - BiochimicaGenotypeMedicineChildhirsutismPolycystic ovaryfemale genital diseases and pregnancy complications030220 oncology & carcinogenesisCohortMolecular MedicineFemalemedicine.symptomAdultHeterozygotecongenital hereditary and neonatal diseases and abnormalitiesAdolescentYoung Adult03 medical and health sciencesHumansCongenital adrenal hyperplasiaMolecular BiologyHeterozygous carrierAdrenal Hyperplasia Congenitalbusiness.industryHyperandrogenismCongenital adrenal hyperplasianutritional and metabolic diseasesHeterozygote advantageCell BiologyOverweightmedicine.diseaseOligomenorrhea17OHProgesterone deficiency030104 developmental biologyMutationSteroid 21-HydroxylaseHyperandrogenismbusinessThe Journal of Steroid Biochemistry and Molecular Biology
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Alterations in Lipid and Carbohydrate Metabolism in Patients with Classic Congenital Adrenal Hyperplasia due to 21-Hydroxylase Deficiency

2009

<i>Background:</i> Classic 21-hydroxylase deficiency (21HD) presents some traits of the metabolic syndrome. <i>Aim:</i> To characterize discrete alterations of lipid and carbohydrate metabolism in children and young adults with classic 21HD, which could predict early atherogenesis. <i>Patients and Methods:</i> Twenty-seven Caucasian patients with classic 21HD (4–31 years); 27 sex-, age- and BMI-matched controls. Clinical parameters, hormonal status and genotype were assessed in all patients. Lipid parameters, including relative (%) and absolute (mg/dl) small-dense low-density lipoproteins subfractions (sd-LDL) were measured in patients and controls. Oral …

AdultBlood GlucoseMalemedicine.medical_specialtyAdolescentEndocrinology Diabetes and MetabolismCarbohydrate metabolismStatistics NonparametricYoung Adultchemistry.chemical_compoundEndocrinologyInternal medicinemedicineHumansInsulinIn patientChildTriglyceridesGlucose tolerance testAdrenal Hyperplasia CongenitalC-Peptidebiologymedicine.diagnostic_testCholesterolCholesterol HDL21-HydroxylaseCholesterol LDLGlucose Tolerance TestHyperplasiaLipid Metabolismmedicine.diseaseCross-Sectional StudiesEndocrinologychemistryChild PreschoolLow-density lipoproteinPediatrics Perinatology and Child Healthbiology.proteinCarbohydrate MetabolismFemaleSteroid 21-HydroxylaseInsulin ResistanceMetabolic syndromeHormone Research in Paediatrics
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The endocrine pattern of late onset adrenal hyperplasia (21-hydroxylase deficiency)

1984

We describe 5 adult women with severe hirsutism due to late onset 21-hydroxylase deficiency. Diagnosis was performed on the finding of high serum 17-hydroxyprogesterone (17OHP) levels with a marked hyperresponse to an ACTH test. The endocrine study showed in most patients a gonadotropin behavior similar to that observed in classical polycystic ovary (PCO) syndrome. Prolactin levels were slightly increased in basal conditions and presented an exaggerated response to TRH stimulation.

AdultHirsutismendocrine systemmedicine.medical_specialtyAdolescentmedicine.drug_classEndocrinology Diabetes and MetabolismLate onsetEndocrinologyAdrenocorticotropic HormoneInternal medicineHydroxyprogesteronesmedicineHumansEndocrine systemGonadal Steroid HormoneshirsutismAdrenal Hyperplasia Congenitalbiologybusiness.industry17-alpha-HydroxyprogesteroneVirilization21-HydroxylaseLuteinizing Hormonemedicine.diseasePolycystic ovaryProlactinProlactinEndocrinologySteroid Hydroxylasesbiology.proteinFemaleSteroid 21-HydroxylaseFollicle Stimulating Hormonemedicine.symptomGonadotropinbusinesshormones hormone substitutes and hormone antagonistsJournal of Endocrinological Investigation
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Reproductive outcome of women with 21-hydroxylase-deficient nonclassic adrenal hyperplasia.

2006

Many women with 21-hydroxylase (21-OH)-deficient nonclassic adrenal hyperplasia (NCAH) carry at least one allele containing a severe mutation of CYP21, and as such are at risk for giving birth to an infant having classic adrenal hyperplasia (CAH). Infants with NCAH typically are asymptomatic at birth, in contrast to those with CAH, but they do develop symptoms of hyperandrogenism later in childhood or as adults. This international multicenter study, conducted both retrospectively and prospectively, was an attempt to determine how often mothers with 21-OH-deficient NCAH bear infants having CAH or NCAH. The 101 women entering the study had a total of 203 pregnancies that could be evaluated. F…

AdultMalePediatricsmedicine.medical_specialtyReferralGenotypeOffspringEndocrinology Diabetes and MetabolismClinical BiochemistryContext (language use)AsymptomaticBiochemistryEndocrinologyPregnancyInternal medicinePrevalenceMedicineHumansProspective StudiesProspective cohort studyAdrenal HyperplasiaRetrospective StudiesPregnancybiologyAdrenal Hyperplasia Congenitalbusiness.industryIncidence (epidemiology)HyperandrogenismBiochemistry (medical)21-HydroxylaseInfant NewbornObstetrics and GynecologyInfantRetrospective cohort studyGeneral MedicineHyperplasiamedicine.diseaseEndocrinologyGlucocorticoid therapyChild Preschoolbiology.proteinFemaleSteroid 21-Hydroxylasemedicine.symptomLive birthbusinessThe Journal of clinical endocrinology and metabolism
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21-Hydroxylase and 11beta-hydroxylase mutations in Romanian patients with classic congenital adrenal hyperplasia.

2005

Congenital adrenal hyperplasia (CAH) comprises autosomal recessive disorders mainly due to defects in the 21-hydroxylase (CYP21) gene.The study aimed to perform molecular characterization in 43 Romanian patients with classical CAH forms diagnosed at the Center for Genetic Diseases of the Pediatric Clinic/University Cluj (38 with 21-hydroxylase deficiency, five with 11beta-hydroxylase deficiency), to determine the frequency of mutations in the CYP21A2 gene and attempt a genotype-phenotype correlation in patients with 21-hydroxylase deficiency.Molecular analysis was performed by direct sequencing of PCR amplified products of the CYP21A2 and CYP11B1 genes.The most frequent mutation in Romanian…

AdultMalemedicine.medical_specialtyAdolescentGenotypeEndocrinology Diabetes and MetabolismClinical BiochemistryContext (language use)Biologymedicine.disease_causeBiochemistryEndocrinologyGene FrequencyInternal medicineGenotypemedicineHumansCongenital adrenal hyperplasiaSteroid 11-beta-hydroxylaseChildAllelesMutationAdrenal Hyperplasia CongenitalRomaniaBiochemistry (medical)Genetic disorder21-HydroxylaseInfantmedicine.diseaseEndocrinologyChild PreschoolMutationbiology.proteinMutation testingSteroid 11-beta-HydroxylaseFemaleSteroid 21-HydroxylaseThe Journal of clinical endocrinology and metabolism
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Refractory Acne and 21-Hydroxylase Deficiency in a Selected Group of Female Patients.

2009

<i>Background:</i> Excessive androgen production, suspected in women when acne is accompanied by hirsutism and menstrual irregularities, may be due to congenital adrenal hyperplasia. This inherited disorder of cortisol biosynthesis is caused in more than 90–95% of all cases by 21-hydroxylase deficiency (21-OHD). The steroid 21-hydroxylase gene <i>(CYP21)</i> has a high degree of variability. <i>Objective:</i> This study was conducted to evaluate <i>CYP21 </i>gene mutations in a selected group of women with papulopustular and comedonal acne refractory to treatment, irregular menses and hirsutism. <i>Methods:</i> 30 out of 61 women e…

Adultmedicine.medical_specialtyAdolescentDrug ResistancePhysiologyDermatologyAdrenocorticotropic hormoneYoung AdultSettore MED/38 - Pediatria Generale E SpecialisticaPapulopustularInternal medicineAcne VulgarismedicineHumansPoint MutationCongenital adrenal hyperplasiaGenetic TestingRefractory acne Excessive androgen production Non-classical 21-hydroxylase deficiency CYP21 gene mutations.AcnehirsutismAdrenal Hyperplasia Congenitalmedicine.diagnostic_testbiologybusiness.industry17-alpha-HydroxyprogesteroneACTH stimulation test21-Hydroxylasemedicine.diseasePolycystic ovaryEndocrinologybiology.proteinFemaleSteroid 21-HydroxylaseHyperandrogenismbusinessPolycystic Ovary Syndrome
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C4 DNA RFLP reference typing report.

1990

One hundred and three individual DNA samples (including 23 families) were studied at the gene level during the reference typing of the fourth component of human complement at the VIth Complement Genetics Workshop in Mainz (1989). All samples were analyzed with the restriction enzyme Taq I and with two DNA probes recognizing the 5' ends of both C4 genes and the two adjacent 21-hydroxylase genes. This RFLP is informative for the number of C4 genes as well as for their respective gene size. We found a high degree of variation regarding the number of C4 genes, i.e. haplotypes with 1-3 structural C4 genes of 16 or 22 kb size. By correlating these haplotypes to the complotypes obtained by protein…

ImmunologyBiologyMajor Histocompatibility Complexchemistry.chemical_compoundHumansTypingDeoxyribonucleases Type II Site-SpecificGeneAllelesGeneticsModels GeneticHybridization probeHaplotypeGenetic VariationComplement C4HematologyDNARestriction enzymeBlotting SouthernchemistryHaplotypesMultilocus sequence typingSteroid 21-HydroxylaseRestriction fragment length polymorphismDNAPolymorphism Restriction Fragment LengthComplement and inflammation
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Major histocompatibility complex (MHC) class III genetics in two Amerindian tribes from southern Brazil: the Kaingang and the Guarani.

1997

Population genetic studies of the major histocompatibility complex (MHC) class III region, comprising C2, BF and C4 phenotypes, and molecular genetic data are rarely available for populations other than Caucasoids. We have investigated three Amerindian populations from Southern Brazil: 131 Kaingang from Ivaí (KIV), 111 Kaingang (KRC) and 100 Guarani (GRC) from Rio das Cobras. Extended MHC haplotypes were derived after standard C2, BF, C4 phenotyping and restriction fragment length polymorphism (RFLP) analysis with TaqI, together with HLA data published previously by segregation analysis. C2 and BF frequencies corresponded to other Amerindian populations. C4B*Q0 frequency was high in the GRC…

MaleTaqIPopulationLocus (genetics)Human leukocyte antigenBiologyMajor Histocompatibility Complexchemistry.chemical_compoundGene FrequencyGeneticsHumanseducationChildGenetics (clinical)Geneticseducation.field_of_studyHistocompatibility TestingIndians South AmericanHaplotypeComplement C4Complement System ProteinsComplement C2Genetic distancechemistryHaplotypesGenetic markerFemaleSteroid 21-HydroxylaseRestriction fragment length polymorphismBrazilPolymorphism Restriction Fragment LengthComplement Factor BHuman genetics
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Ovarian and Adrenal Hyperandrogenism

2007

Because in normal women androgens are secreted in almost equal quantities by both adrenals and ovaries, for many years many studies have tried to distinguish the source of androgen excess. However, in the last 10-15 years, the diagnoses of ovarian or adrenal hyperandrogenism have almost disappeared. This is due to the lack of specificity of dynamic tests as well as to the emphasis given on clinical information and ovarian sonography for the diagnosis of hyperandrogenic syndromes. However, determination of the source of increased androgens may still be useful for improving the classification and the understanding of androgen excess disorders. The aim of this review is to examine the source o…

medicine.medical_specialtymedicine.drug_classidiopathic hyperandrogenismOvaryurologic and male genital diseasesAndrogen ExcessGeneral Biochemistry Genetics and Molecular Biologychemistry.chemical_compoundDehydroepiandrosterone sulfateNCAHHistory and Philosophy of ScienceInternal medicineAdrenal GlandsPCOSmedicineHyperinsulinemiaHumansbusiness.industryGeneral NeuroscienceOvaryHyperandrogenismnonclassic 21-hydroxylase deficiencymedicine.diseaseAndrogenPolycystic ovaryAndrogen secretionEndocrinologymedicine.anatomical_structurechemistryFemaleSteroid 21-Hydroxylaseandrogen excessHyperandrogenismbusinessPolycystic Ovary SyndromeAnnals of the New York Academy of Sciences
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